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OCR: mia result Two major genetic forms of hyperlipida from increased VLDL - input familial lined hypertriglyceridaemia and familial com the VLDL hyperlipidaemia. In the former conditio particles that enter plasma are large and they are triglyceride-enriched, while in the latter primary smaller and less rich in triglycerides. Th defect in familial hypertriglyceridaemia elated to overproduction of cholic acid, which is n familial increased hepatic triglyceride synthesis. unknown, combined hyperlipidaemia this defect is although the disorder is characterised by is overproduction of apo B (Fig. 6.11): apo y to overproduced in VLDL, leading primari İL, leading hypertriglyceridaemia; occasionally in L primarily to hypercholesterolaemia; and Ing to occasionally in both VLDL and LDL, lead riglyceride ...